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An HDAC3-PROX1 corepressor module acts on HNF4α to control hepatic triglycerides
The histone deacetylase HDAC3 is a critical mediator of hepatic lipid metabolism, and liver-specific deletion of HDAC3 leads to fatty liver. To elucidate the underlying mechanism, here we report a method of cross-linking followed by mass spectrometry to define a high-confidence HDAC3 interactome in...
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| Publicado no: | Nat Commun |
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| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5601916/ https://ncbi.nlm.nih.gov/pubmed/28916805 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-017-00772-5 |
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