Rapamycin attenuates BAFF-extended proliferation and survival via disruption of mTORC1/2 signaling in normal and neoplastic B-lymphoid cells

Podobne zapisy

• Rapamycin inhibits B-cell activating factor (BAFF)-stimulated cell proliferation and survival by suppressing Ca(2+)-CaMKII-dependent PTEN/Akt-Erk1/2 signaling pathway in normal and neoplastic B-lymphoid cells
  od: Zeng, Qingyu, i wsp.
  Wydane: (2020)
• Rapamycin prevents cadmium-induced neuronal cell death via targeting both mTORC1 and mTORC2 pathways
  od: Xu, Chong, i wsp.
  Wydane: (2015)
• BAFF inhibits autophagy promoting cell proliferation and survival by activating Ca(2+)-CaMKII-dependent Akt/mTOR signaling pathway in normal and neoplastic B-lymphoi d cells
  od: Dong, Xiaoqing, i wsp.
  Wydane: (2018)
• IL-2, IL-4, IFN-γ or TNF-α enhances BAFF-stimulated cell viability and survival by activating Erk1/2 and S6K1 pathways in neoplastic B-lymphoid cells
  od: Gui, Lin, i wsp.
  Wydane: (2016)
• Rapamycin inhibits mSin1 phosphorylation independently of mTORC1 and mTORC2
  od: Luo, Yan, i wsp.
  Wydane: (2015)