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The Fanconi Anemia Protein Interaction Network: Casting A Wide Net
It has long been hypothesized that a defect in the repair of damaged DNA is central to the etiology of Fanconi anemia (FA). Indeed, an increased sensitivity of FA patient-derived cells to the lethal effects of various forms of DNA damaging agents was described over three decades ago [1–3]. Furthermo...
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| Publié dans: | Mutat Res |
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| Auteurs principaux: | , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2008
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5578810/ https://ncbi.nlm.nih.gov/pubmed/19101576 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mrfmmm.2008.11.018 |
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