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Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules
Aberrant activation of with no lysine (WNK) kinases causes familial hyperkalemic hypertension (FHHt). Thiazide diuretics treat the disease, fostering the view that hyperactivation of the thiazide-sensitive sodium-chloride cotransporter (NCC) in the distal convoluted tubule (DCT) is solely responsibl...
Gorde:
| Argitaratua izan da: | J Am Soc Nephrol |
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| Egile Nagusiak: | , , , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
American Society of Nephrology
2017
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5576927/ https://ncbi.nlm.nih.gov/pubmed/28442491 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2016090948 |
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