ロード中...
RECQL4 promotes DNA end resection in repair of DNA double-strand breaks
The RecQ helicase RECQL4, mutated in Rothmund-Thomson syndrome, regulates genome stability, aging and cancer. Here, we identify a crucial role for RECQL4 in DNA end resection, which is the initial and an essential step of homologous recombination (HR)-dependent DNA double-strand break repair (DSBR)....
保存先:
| 出版年: | Cell Rep |
|---|---|
| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2016
|
| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5576896/ https://ncbi.nlm.nih.gov/pubmed/27320928 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2016.05.079 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|