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RECQL4 promotes DNA end resection in repair of DNA double-strand breaks
The RecQ helicase RECQL4, mutated in Rothmund-Thomson syndrome, regulates genome stability, aging and cancer. Here, we identify a crucial role for RECQL4 in DNA end resection, which is the initial and an essential step of homologous recombination (HR)-dependent DNA double-strand break repair (DSBR)....
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| Publicado no: | Cell Rep |
|---|---|
| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5576896/ https://ncbi.nlm.nih.gov/pubmed/27320928 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2016.05.079 |
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