Intraneuronal accumulation of Aβ(42) induces age-dependent slowing of neuronal transmission in Drosophila

Beta amyloid (Aβ(42))-induced dysfunction and loss of synapses are believed to be major underlying mechanisms for the progressive loss of learning and memory abilities in Alzheimer’s disease (AD). The vast majority of investigations on AD-related synaptic impairment focus on synaptic plasticity, esp...

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Detalhes bibliográficos
Publicado no:Neurosci Bull
Main Authors: Lin, Jing-Ya, Wang, Wen-An, Zhang, Xiao, Liu, Hai-Yan, Zhao, Xiao-Liang, Huang, Fu-De
Formato: Artigo
Idioma:Inglês
Publicado em: Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences 2014
Assuntos:
Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5562655/
https://ncbi.nlm.nih.gov/pubmed/24733651
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12264-013-1409-9
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