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Concurrent activation of β(2)-adrenergic receptor and blockage of GPR55 disrupts pro-oncogenic signaling in glioma cells

Activation of β(2)-adrenergic receptor (β(2)AR) and deorphanized GPR55 has been shown to modulate cancer growth in diverse tumor types in vitro and in xenograft models in vivo. (R,R′)-4′-methoxy-1-naphthylfenoterol [(R,R′)-MNF] is a bivalent compound that agonizes β(2)AR but inhibits GPR55-mediated...

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Detalhes bibliográficos
Publicado no:Cell Signal
Main Authors: Wnorowski, Artur, Such, Justyna, Paul, Rajib K., Wersto, Robert P., Indig, Fred E., Jozwiak, Krzysztof, Bernier, Michel, Wainer, Irving W.
Formato: Artigo
Idioma:Inglês
Publicado em: 2017
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5512002/
https://ncbi.nlm.nih.gov/pubmed/28495590
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2017.05.006
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