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Differential Protein Acetylation Assists Import of Excess SOD2 into Mitochondria and Mediates SOD2 Aggregation Associated with Cardiac Hypertrophy in the Murine SOD2-tg Heart
SOD2 is the primary antioxidant enzyme neutralizing (•)O(2)(−) in mitochondria. Cardiac-specific SOD2 overexpression (SOD2-tg) induces supernormal function and cardiac hypertrophy in the mouse heart. However, the reductive stress imposed by SOD2 overexpression results in protein aggregation of SOD2...
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| 出版年: | Free Radic Biol Med |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5487267/ https://ncbi.nlm.nih.gov/pubmed/28433661 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2017.04.022 |
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