Excess NO Predisposes Mitochondrial Succinate-Cytochrome c Reductase to Produce Hydroxyl Radical(†)

Mitochondria–derived oxygen free radical(s) are important mediators of oxidative cellular injury. It is widely hypothesized that excess NO enhances O(2)(•−) generated by mitochondria under certain pathological conditions. In the mitochondrial electron transport chain, succinate-cytochrome c reductas...

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Hlavní autoři: Chen, Jingfeng, Chen, Chwen-Lih, Alevriadou, B. Rita, Zweier, Jay L., Chen, Yeong-Renn
Médium: Artigo
Jazyk:Inglês
Vydáno: 2011
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3698227/
https://ncbi.nlm.nih.gov/pubmed/21406178
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbabio.2011.03.001
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