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Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex III
Recent evidence has implicated succinate-driven reverse electron transport (RET) through complex I as a major source of damaging reactive oxygen species (ROS) underlying reperfusion injury after prolonged cardiac ischemia. However, this explanation may be incomplete, because RET on reperfusion is se...
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| Publicado en: | J Biol Chem |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
American Society for Biochemistry and Molecular Biology
2017
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5473241/ https://ncbi.nlm.nih.gov/pubmed/28450391 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M116.768317 |
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