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MuRF1 mono-ubiquitinates TRα to inhibit T3-induced cardiac hypertrophy in vivo
Thyroid hormone (TH) is recognized for its role in cellular metabolism and growth and participates in homeostasis of the heart. T3 activates pro-survival pathways including Akt and mTOR. Treatment with T3 after myocardial infarction is cardioprotective and promotes elements of physiological hypertro...
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| Veröffentlicht in: | J Mol Endocrinol |
|---|---|
| Hauptverfasser: | , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2016
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5453669/ https://ncbi.nlm.nih.gov/pubmed/26862156 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/JME-15-0283 |
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