Smooth muscle cell-specific deletion of Col15a1 unexpectedly leads to impaired development of advanced atherosclerotic lesions

Atherosclerotic plaque rupture with subsequent embolic events is a major cause of sudden death from myocardial infarction or stroke. Although smooth muscle cells (SMCs) produce and respond to collagens in vitro, there is no direct evidence in vivo that SMCs are a crucial source of collagens and that...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Am J Physiol Heart Circ Physiol
Päätekijät: Durgin, Brittany G., Cherepanova, Olga A., Gomez, Delphine, Karaoli, Themistoclis, Alencar, Gabriel F., Butcher, Joshua T., Zhou, Yu-Qing, Bendeck, Michelle P., Isakson, Brant E., Owens, Gary K., Connelly, Jessica J.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2017
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5451587/
https://ncbi.nlm.nih.gov/pubmed/28283548
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00029.2017
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