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A novel ALS-associated variant in UBQLN4 regulates motor axon morphogenesis
The etiological underpinnings of amyotrophic lateral sclerosis (ALS) are complex and incompletely understood, although contributions to pathogenesis by regulators of proteolytic pathways have become increasingly apparent. Here, we present a novel variant in UBQLN4 that is associated with ALS and sho...
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| Publicado no: | eLife |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
eLife Sciences Publications, Ltd
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5451210/ https://ncbi.nlm.nih.gov/pubmed/28463112 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.25453 |
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