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Functional Interaction of H2AX, NBS1, and p53 in ATM-Dependent DNA Damage Responses and Tumor Suppression

Ataxia-telangiectasia (A-T) mutated (ATM) kinase signals all three cell cycle checkpoints after DNA double-stranded break (DSB) damage. H2AX, NBS1, and p53 are substrates of ATM kinase and are involved in ATM-dependent DNA damage responses. We show here that H2AX is dispensable for the activation of...

Deskribapen osoa

Gorde:
Argitaratua izan da:Mol Cell Biol
Egile Nagusiak: Kang, Jian, Ferguson, David, Song, Hoseok, Bassing, Craig, Eckersdorff, Mark, Alt, Frederick W., Xu, Yang
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Taylor & Francis 2005
Gaiak:
Signal Transduction
Sarrera elektronikoa:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC543410/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15632067/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.25.2.661-670.2005
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