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Phosphorylation of Hdmx mediates its Hdm2- and ATM-dependent degradation in response to DNA damage
Maintenance of genomic stability depends on the DNA damage response, an extensive signaling network that is activated by DNA lesions such as double-strand breaks (DSBs). The primary activator of the mammalian DSB response is the nuclear protein kinase ataxia–telangiectasia, mutated (ATM), which phos...
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| Главные авторы: | , , , , , , , , , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
National Academy of Sciences
2005
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC555986/ https://ncbi.nlm.nih.gov/pubmed/15788536 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0408595102 |
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