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Metabolic fate of glucose and candidate signaling and excess-fuel detoxification pathways in pancreatic β-cells
Glucose metabolism promotes insulin secretion in β-cells via metabolic coupling factors that are incompletely defined. Moreover, chronically elevated glucose causes β-cell dysfunction, but little is known about how cells handle excess fuels to avoid toxicity. Here we sought to determine which among...
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| Pubblicato in: | J Biol Chem |
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| Autori principali: | , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Biochemistry and Molecular Biology
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5418042/ https://ncbi.nlm.nih.gov/pubmed/28280244 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M116.763060 |
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