C-terminal truncated hepatitis B virus X protein regulates tumorigenicity, self-renewal and drug resistance via STAT3/Nanog signaling pathway

Hepatitis B virus (HBV) is a major risk factor of chronic liver disease and hepatocellular carcinoma (HCC). Random integration of HBV DNA into the host genome is frequent in HCC leading to truncation of the HBV DNA, particularly at the C-terminal end of the HBV X protein (HBx). C-terminally truncate...

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Detalles Bibliográficos
Publicado en:Oncotarget
Main Authors: Ching, Rachel Hiu Ha, Sze, Karen Man Fong, Lau, Eunice Yuen Ting, Chiu, Yung-Tuen, Lee, Joyce Man Fong, Ng, Irene Oi Lin, Lee, Terence Kin Wah
Formato: Artigo
Idioma:Inglês
Publicado: Impact Journals LLC 2017
Assuntos:
Research Paper
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC5410322/
https://ncbi.nlm.nih.gov/pubmed/28186991
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.15183
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