C-terminal truncated hepatitis B virus X protein regulates tumorigenicity, self-renewal and drug resistance via STAT3/Nanog signaling pathway

Hepatitis B virus (HBV) is a major risk factor of chronic liver disease and hepatocellular carcinoma (HCC). Random integration of HBV DNA into the host genome is frequent in HCC leading to truncation of the HBV DNA, particularly at the C-terminal end of the HBV X protein (HBx). C-terminally truncate...

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Bibliographische Detailangaben
Veröffentlicht in:Oncotarget
Hauptverfasser: Ching, Rachel Hiu Ha, Sze, Karen Man Fong, Lau, Eunice Yuen Ting, Chiu, Yung-Tuen, Lee, Joyce Man Fong, Ng, Irene Oi Lin, Lee, Terence Kin Wah
Format: Artigo
Sprache:Inglês
Veröffentlicht: Impact Journals LLC 2017
Schlagworte:
Research Paper
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5410322/
https://ncbi.nlm.nih.gov/pubmed/28186991
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.15183
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