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Low pH(o) boosts burst firing and catecholamine release by blocking TASK‐1 and BK channels while preserving Cav1 channels in mouse chromaffin cells
KEY POINTS: Mouse chromaffin cells (MCCs) generate spontaneous burst‐firing that causes large increases of Ca(2+)‐dependent catecholamine release, and is thus a key mechanism for regulating the functions of MCCs. With the aim to uncover a physiological role for burst‐firing we investigated the effec...
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| Publicat a: | J Physiol |
|---|---|
| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
John Wiley and Sons Inc.
2017
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5390891/ https://ncbi.nlm.nih.gov/pubmed/28026020 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP273735 |
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