Inactivation of ID4 promotes a CRPC phenotype with constitutive AR activation through FKBP52

Castration‐resistant prostate cancer (CRPC) is the emergence of prostate cancer cells that have adapted to the androgen‐depleted environment of the prostate. In recent years, targeting multiple chaperones and co‐chaperones (e.g., Hsp27, FKBP52) that promote androgen receptor (AR) signaling and/or no...

詳細記述

保存先:
書誌詳細
出版年:Mol Oncol
主要な著者: Joshi, Jugal Bharat, Patel, Divya, Morton, Derrick J., Sharma, Pankaj, Zou, Jin, Hewa Bostanthirige, Dhanushka, Gorantla, Yamini, Nagappan, Peri, Komaragiri, Shravan Kumar, Sivils, Jeffrey C., Xie, Huan, Palaniappan, Ravi, Wang, Guangdi, Cox, Marc B., Chaudhary, Jaideep
フォーマット: Artigo
言語:Inglês
出版事項: John Wiley and Sons Inc. 2017
主題:
Research Articles
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC5378613/
https://ncbi.nlm.nih.gov/pubmed/28252832
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/1878-0261.12028
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