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Inhibition of androgen receptor by decoy molecules delays progression to castration-recurrent prostate cancer
Androgen receptor (AR) is a member of the steroid receptor family and a therapeutic target for all stages of prostate cancer. AR is activated by ligand binding within its C-terminus ligand-binding domain (LBD). Here we show that overexpression of the AR NTD to generate decoy molecules inhibited both...
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| 出版年: | PLoS One |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Public Library of Science
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5357013/ https://ncbi.nlm.nih.gov/pubmed/28306720 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0174134 |
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