Src promotes castration-recurrent prostate cancer through androgen receptor-dependent canonical and non-canonical transcriptional signatures

Progression of prostate cancer (PC) to castration-recurrent growth (CRPC) remains dependent on sustained expression and transcriptional activity of the androgen receptor (AR). A major mechanism contributing to CRPC progression is through the direct phosphorylation and activation of AR by Src-family...

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Foilsithe in:Oncotarget
Main Authors: Chattopadhyay, Indranil, Wang, Jianmin, Qin, Maochun, Gao, Lingqiu, Holtz, Renae, Vessella, Robert L., Leach, Robert W., Gelman, Irwin H.
Formáid: Artigo
Teanga:Inglês
Foilsithe: Impact Journals LLC 2016
Ábhair:
Research Paper
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5354662/
https://ncbi.nlm.nih.gov/pubmed/28055971
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.14401
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