Acidosis Activates Endoplasmic Reticulum Stress Pathways through GPR4 in Human Vascular Endothelial Cells

Lignende værker

• Activation of GPR4 by Acidosis Increases Endothelial Cell Adhesion through the cAMP/Epac Pathway
  af: Chen, Aishe, et al.
  Udgivet: (2011)
• Acidosis Activation of the Proton-Sensing GPR4 Receptor Stimulates Vascular Endothelial Cell Inflammatory Responses Revealed by Transcriptome Analysis
  af: Dong, Lixue, et al.
  Udgivet: (2013)
• Mechanism of endoplasmic reticulum stress-induced vascular endothelial dysfunction
  af: Galán, Maria, et al.
  Udgivet: (2014)
• High expression of active ATF6 aggravates endoplasmic reticulum stress-induced vascular endothelial cell apoptosis through the mitochondrial apoptotic pathway
  af: Huang, Jingyong, et al.
  Udgivet: (2018)
• The Proton-Sensing GPR4 Receptor Regulates Paracellular Gap Formation and Permeability of Vascular Endothelial Cells
  af: Elizabeth A. Krewson, et al.
  Udgivet: (2020-02-01)