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Protection from cytosolic prion protein toxicity by modulation of protein translocation

Failure to promptly dispose of undesirable proteins is associated with numerous diseases. In the case of cellular prion protein (PrP), inhibition of the proteasome pathway can generate a highly aggregation-prone, cytotoxic form of PrP implicated in neurodegeneration. However, the predominant mechani...

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Detalhes bibliográficos
Main Authors: Rane, Neena S, Yonkovich, Jesse L, Hegde, Ramanujan S
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2004
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC533048/
https://ncbi.nlm.nih.gov/pubmed/15526034
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7600462
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