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β-cell insulin receptor deficiency during in utero development induces an islet compensatory overgrowth response

The presence of insulin receptor (IR) on β-cells suggests that insulin has an autocrine/paracrine role in the regulation of β-cell function. It has previously been reported that the β-cell specific loss of IR (βIRKO) leads to the development of impaired glycemic regulation and β-cell death in mice....

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Detalhes bibliográficos
Publicado no:Oncotarget
Main Authors: Trinder, Mark, Zhou, Liangyi, Oakie, Amanda, Riopel, Matthew, Wang, Rennian
Formato: Artigo
Idioma:Inglês
Publicado em: Impact Journals LLC 2016
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5216695/
https://ncbi.nlm.nih.gov/pubmed/27384998
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.10342
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