MyD88 adapter-like (Mal) is phosphorylated by Bruton's tyrosine kinase during TLR2 and TLR4 signal transduction.

Lignende værker

• IRAK1 and IRAK4 promote phosphorylation, ubiquitination, and degradation of MyD88 adaptor-like (Mal).
  af: Dunne, Aisling, et al.
  Udgivet: (2016)
• IRAK1 and IRAK4 Promote Phosphorylation, Ubiquitination, and Degradation of MyD88 Adaptor-like (Mal)
  af: Dunne, Aisling, et al.
  Udgivet: (2010)
• TYROSINE PHOSPHORYLATION OF MYD88 ADAPTER-LIKE (MAL) IS CRITICAL FOR SIGNAL TRANSDUCTION AND BLOCKED IN ENDOTOXIN TOLERANCE
  af: Piao, Wenji, et al.
  Udgivet: (2007)
• A TIR Domain Variant of MyD88 Adapter-like (Mal)/TIRAP Results in Loss of MyD88 Binding and Reduced TLR2/TLR4 Signaling
  af: Nagpal, Kamalpreet, et al.
  Udgivet: (2009)
• Structural basis of TIR-domain assembly formation in MyD88/MAL-dependent TLR4 signaling
  af: Ve, Thomas, et al.
  Udgivet: (2017)