Association of Amyloid Pathology With Myelin Alteration in Preclinical Alzheimer Disease

IMPORTANCE: The accumulation of aggregated β-amyloid and tau proteins into plaques and tangles is a central feature of Alzheimer disease (AD). While plaque and tangle accumulation likely contributes to neuron and synapse loss, disease-related changes to oligodendrocytes and myelin are also suspected...

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Опубликовано в: :JAMA Neurol
Главные авторы: Dean, Douglas C., Hurley, Samuel A., Kecskemeti, Steven R., O’Grady, J. Patrick, Canda, Cristybelle, Davenport-Sis, Nancy J., Carlsson, Cynthia M., Zetterberg, Henrik, Blennow, Kaj, Asthana, Sanjay, Sager, Mark A., Johnson, Sterling C., Alexander, Andrew L., Bendlin, Barbara B.
Формат: Artigo
Язык:Inglês
Опубликовано: 2017
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Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5195903/
https://ncbi.nlm.nih.gov/pubmed/27842175
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1001/jamaneurol.2016.3232
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