Artemis Is a Phosphorylation Target of ATM and ATR and Is Involved in the G(2)/M DNA Damage Checkpoint Response
Mutations in Artemis in both humans and mice result in severe combined immunodeficiency due to a defect in V(D)J recombination. In addition, Artemis mutants are radiosensitive and chromosomally unstable, which has been attributed to a defect in nonhomologous end joining (NHEJ). We show here, however...
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| Wydane w: | Mol Cell Biol |
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| Główni autorzy: | , , , , , |
| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
Taylor & Francis
2004
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC517881/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15456891/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.24.20.9207-9220.2004 |
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