Artemis Is a Phosphorylation Target of ATM and ATR and Is Involved in the G(2)/M DNA Damage Checkpoint Response
Mutations in Artemis in both humans and mice result in severe combined immunodeficiency due to a defect in V(D)J recombination. In addition, Artemis mutants are radiosensitive and chromosomally unstable, which has been attributed to a defect in nonhomologous end joining (NHEJ). We show here, however...
Tallennettuna:
| Julkaisussa: | Mol Cell Biol |
|---|---|
| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Taylor & Francis
2004
|
| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC517881/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15456891/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.24.20.9207-9220.2004 |
| Tagit: |
Ei tageja, Lisää ensimmäinen tagi!
|
