Protein Kinases C and D Mediate Agonist-Dependent Cardiac Hypertrophy through Nuclear Export of Histone Deacetylase 5

A variety of stress signals stimulate cardiac myocytes to undergo hypertrophy. Persistent cardiac hypertrophy is associated with elevated risk for the development of heart failure. Recently, we showed that class II histone deacetylases (HDACs) suppress cardiac hypertrophy and that stress signals neu...

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Dettagli Bibliografici
Pubblicato in:Mol Cell Biol
Autori principali: Vega, Rick B., Harrison, Brooke C., Meadows, Eric, Roberts, Charles R., Papst, Philip J., Olson, Eric N., McKinsey, Timothy A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Taylor & Francis 2004
Soggetti:
Transcriptional Regulation
Accesso online:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC516754/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15367659/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.24.19.8374-8385.2004
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