Protein Kinases C and D Mediate Agonist-Dependent Cardiac Hypertrophy through Nuclear Export of Histone Deacetylase 5

A variety of stress signals stimulate cardiac myocytes to undergo hypertrophy. Persistent cardiac hypertrophy is associated with elevated risk for the development of heart failure. Recently, we showed that class II histone deacetylases (HDACs) suppress cardiac hypertrophy and that stress signals neu...

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Bibliografiske detaljer
Udgivet i:Mol Cell Biol
Main Authors: Vega, Rick B., Harrison, Brooke C., Meadows, Eric, Roberts, Charles R., Papst, Philip J., Olson, Eric N., McKinsey, Timothy A.
Format: Artigo
Sprog:Inglês
Udgivet: Taylor & Francis 2004
Fag:
Transcriptional Regulation
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC516754/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15367659/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.24.19.8374-8385.2004
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