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BLM and the FANC proteins collaborate in a common pathway in response to stalled replication forks
Fanconi anaemia (FA) and Bloom syndrome (BS) are autosomal recessive diseases characterised by chromosome fragility and cancer proneness. Here, we report that BLM and the FA pathway are activated in response to both crosslinked DNA and replication fork stall. We provide evidence that BLM and FANCD2...
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| 出版年: | EMBO J |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group
2004
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC514912/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15257300/ https://ncbi.nlm.nih.govhttps://doi.org/10.1038/sj.emboj.7600277 |
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