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Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart

Previous results from our laboratory showed that phosphorylation of ryanodine receptor 2 (RyR2) by Ca(2+) calmodulin-dependent kinase II (CaMKII) was a critical but not the unique event responsible for the production of reperfusion-induced arrhythmogenesis, suggesting the existence of other mechanis...

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Detalhes bibliográficos
Publicado no:Am J Physiol Heart Circ Physiol
Main Authors: Becerra, Romina, Román, Bárbara, Di Carlo, Mariano N., Mariangelo, Juan Ignacio, Salas, Margarita, Sanchez, Gina, Donoso, Paulina, Schinella, Guillermo R., Vittone, Leticia, Wehrens, Xander H., Mundiña-Weilenmann, Cecilia, Said, Matilde
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2016
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5142185/
https://ncbi.nlm.nih.gov/pubmed/27422983
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00142.2016
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