Pancreatic α-cell hyperplasia and hyperglucagonemia due to a glucagon receptor splice mutation

Glucagon stimulates hepatic glucose production by activating specific glucagon receptors in the liver, which in turn increase hepatic glycogenolysis as well as gluconeogenesis and ureagenesis from amino acids. Conversely, glucagon secretion is regulated by concentrations of glucose and amino acids....

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Detalles Bibliográficos
Publicado en:Endocrinol Diabetes Metab Case Rep
Main Authors: Larger, Etienne, Wewer Albrechtsen, Nicolai J, Hansen, Lars H, Gelling, Richard W, Capeau, Jacqueline, Deacon, Carolyn F, Madsen, Ole D, Yakushiji, Fumiatsu, De Meyts, Pierre, Holst, Jens J, Nishimura, Erica
Formato: Artigo
Idioma:Inglês
Publicado: Bioscientifica Ltd 2016
Assuntos:
Insight into Disease Pathogenesis or Mechanism of Therapy
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118975/
https://ncbi.nlm.nih.gov/pubmed/27933176
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/EDM-16-0081
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