Pancreatic α-cell hyperplasia and hyperglucagonemia due to a glucagon receptor splice mutation

Glucagon stimulates hepatic glucose production by activating specific glucagon receptors in the liver, which in turn increase hepatic glycogenolysis as well as gluconeogenesis and ureagenesis from amino acids. Conversely, glucagon secretion is regulated by concentrations of glucose and amino acids....

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Endocrinol Diabetes Metab Case Rep
Egile Nagusiak: Larger, Etienne, Wewer Albrechtsen, Nicolai J, Hansen, Lars H, Gelling, Richard W, Capeau, Jacqueline, Deacon, Carolyn F, Madsen, Ole D, Yakushiji, Fumiatsu, De Meyts, Pierre, Holst, Jens J, Nishimura, Erica
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Bioscientifica Ltd 2016
Gaiak:
Insight into Disease Pathogenesis or Mechanism of Therapy
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118975/
https://ncbi.nlm.nih.gov/pubmed/27933176
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/EDM-16-0081
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