Pancreatic α-cell hyperplasia and hyperglucagonemia due to a glucagon receptor splice mutation

Glucagon stimulates hepatic glucose production by activating specific glucagon receptors in the liver, which in turn increase hepatic glycogenolysis as well as gluconeogenesis and ureagenesis from amino acids. Conversely, glucagon secretion is regulated by concentrations of glucose and amino acids....

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Vydáno v:Endocrinol Diabetes Metab Case Rep
Hlavní autoři: Larger, Etienne, Wewer Albrechtsen, Nicolai J, Hansen, Lars H, Gelling, Richard W, Capeau, Jacqueline, Deacon, Carolyn F, Madsen, Ole D, Yakushiji, Fumiatsu, De Meyts, Pierre, Holst, Jens J, Nishimura, Erica
Médium: Artigo
Jazyk:Inglês
Vydáno: Bioscientifica Ltd 2016
Témata:
Insight into Disease Pathogenesis or Mechanism of Therapy
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118975/
https://ncbi.nlm.nih.gov/pubmed/27933176
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/EDM-16-0081
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