Pancreatic α-cell hyperplasia and hyperglucagonemia due to a glucagon receptor splice mutation

Glucagon stimulates hepatic glucose production by activating specific glucagon receptors in the liver, which in turn increase hepatic glycogenolysis as well as gluconeogenesis and ureagenesis from amino acids. Conversely, glucagon secretion is regulated by concentrations of glucose and amino acids....

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Dades bibliogràfiques
Publicat a:Endocrinol Diabetes Metab Case Rep
Autors principals: Larger, Etienne, Wewer Albrechtsen, Nicolai J, Hansen, Lars H, Gelling, Richard W, Capeau, Jacqueline, Deacon, Carolyn F, Madsen, Ole D, Yakushiji, Fumiatsu, De Meyts, Pierre, Holst, Jens J, Nishimura, Erica
Format: Artigo
Idioma:Inglês
Publicat: Bioscientifica Ltd 2016
Matèries:
Insight into Disease Pathogenesis or Mechanism of Therapy
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5118975/
https://ncbi.nlm.nih.gov/pubmed/27933176
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/EDM-16-0081
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