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AKT activation controls cell survival in response to HDAC6 inhibition
HDAC6 is emerging as an important therapeutic target for cancer. We investigated mechanisms responsible for survival of tumor cells treated with a HDAC6 inhibitor. Expression of the 20 000 genes examined did not change following HDAC6 treatment in vivo. We found that HDAC6 inhibition led to an incre...
שמור ב:
| הוצא לאור ב: | Cell Death Dis |
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| Main Authors: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Nature Publishing Group
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5108334/ https://ncbi.nlm.nih.gov/pubmed/27362804 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2016.180 |
| תגים: |
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