Interferon-β (IFN-β) facilitates neuroantigen-dependent induction of CD25(+) FOXP3(+) regulatory T cells that suppress experimental autoimmune encephalomyelitis (EAE)()

Antzeko izenburuak

• A GMCSF-neuroantigen fusion protein is a potent tolerogen in experimental autoimmune encephalomyelitis (EAE) that is associated with efficient targeting of neuroantigen to APC
  nork: Blanchfield, J. Lori, et al.
  Argitaratua: (2010)
• GM-CSF-neuroantigen fusion proteins reverse experimental autoimmune encephalomyelitis and mediate tolerogenic activity in adjuvant-primed environments: association with inflammation-dependent, inhibitory antigen presentation()
  nork: Islam, S.M. Touhidul, et al.
  Argitaratua: (2014)
• A GM-CSF-neuroantigen tolerogenic vaccine elicits inefficient antigen recognition events below the CD40L triggering threshold to expand CD4(+) CD25(+) FOXP3(+) Tregs that inhibit experimental autoimmune encephalomyelitis (EAE)
  nork: Moorman, Cody D., et al.
  Argitaratua: (2020)
• Depletion of CD4(+) CD25(+) regulatory T cells confers susceptibility to experimental autoimmune encephalomyelitis (EAE) in GM-CSF-deficient Csf2(−/−) mice
  nork: Ghosh, Debjani, et al.
  Argitaratua: (2016)
• Partial CD25 Antagonism Enables Dominance of Antigen-Inducible CD25(high) FOXP3(+) Regulatory T Cells As a Basis for a Regulatory T Cell-Based Adoptive Immunotherapy
  nork: Wilkinson, Daniel S., et al.
  Argitaratua: (2017)