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Synaptopodin Limits TRPC6 Podocyte Surface Expression and Attenuates Proteinuria
Gain-of-function mutations of classic transient receptor potential channel 6 (TRPC6) were identified in familial FSGS, and increased expression of wild-type TRPC6 in glomeruli is observed in several human acquired proteinuric diseases. Synaptopodin, an actin binding protein that is important in main...
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| Pubblicato in: | J Am Soc Nephrol |
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| Autori principali: | , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society of Nephrology
2016
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5084886/ https://ncbi.nlm.nih.gov/pubmed/27020855 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015080896 |
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