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Amyloid-β effects on synapses and memory require AMPA receptor subunit GluA3
Amyloid-β (Aβ) is a prime suspect for causing cognitive deficits during the early phases of Alzheimer’s disease (AD). Experiments in AD mouse models have shown that soluble oligomeric clusters of Aβ degrade synapses and impair memory formation. We show that all Aβ-driven effects measured in these mi...
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| Publicado en: | Proc Natl Acad Sci U S A |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
National Academy of Sciences
2016
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5081598/ https://ncbi.nlm.nih.gov/pubmed/27708157 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1614249113 |
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