Amyloid-β effects on synapses and memory require AMPA receptor subunit GluA3

Amyloid-β (Aβ) is a prime suspect for causing cognitive deficits during the early phases of Alzheimer’s disease (AD). Experiments in AD mouse models have shown that soluble oligomeric clusters of Aβ degrade synapses and impair memory formation. We show that all Aβ-driven effects measured in these mi...

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Detaylı Bibliyografya
Yayımlandı:Proc Natl Acad Sci U S A
Asıl Yazarlar: Reinders, Niels R., Pao, Yvonne, Renner, Maria C., da Silva-Matos, Carla M., Lodder, Tessa R., Malinow, Roberto, Kessels, Helmut W.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: National Academy of Sciences 2016
Konular:
PNAS Plus
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5081598/
https://ncbi.nlm.nih.gov/pubmed/27708157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1614249113
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