ロード中...
Amyloid-β effects on synapses and memory require AMPA receptor subunit GluA3
Amyloid-β (Aβ) is a prime suspect for causing cognitive deficits during the early phases of Alzheimer’s disease (AD). Experiments in AD mouse models have shown that soluble oligomeric clusters of Aβ degrade synapses and impair memory formation. We show that all Aβ-driven effects measured in these mi...
保存先:
| 出版年: | Proc Natl Acad Sci U S A |
|---|---|
| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
National Academy of Sciences
2016
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5081598/ https://ncbi.nlm.nih.gov/pubmed/27708157 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1614249113 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|