DUSP1 Maintains IRF1 and Leads to Increased Expression of IRF1-dependent Genes: A MECHANISM PROMOTING GLUCOCORTICOID INSENSITIVITY

Similar Items

• Negative Feed-forward Control of Tumor Necrosis Factor (TNF) by Tristetraprolin (ZFP36) Is Limited by the Mitogen-activated Protein Kinase Phosphatase, Dual-specificity Phosphatase 1 (DUSP1): IMPLICATIONS FOR REGULATION BY GLUCOCORTICOIDS
  od: Shah, Suharsh, i dr.
  Izdano: (2016)
• Roles for the Mitogen-activated Protein Kinase (MAPK) Phosphatase, DUSP1, in Feedback Control of Inflammatory Gene Expression and Repression by Dexamethasone
  od: Shah, Suharsh, i dr.
  Izdano: (2014)
• Glucocorticoid and cytokine crosstalk: Feedback, feedforward, and co-regulatory interactions determine repression or resistance
  od: Newton, Robert, i dr.
  Izdano: (2017)
• Context-dependent Cooperation between Nuclear Factor κB (NF-κB) and the Glucocorticoid Receptor at a TNFAIP3 Intronic Enhancer: A MECHANISM TO MAINTAIN NEGATIVE FEEDBACK CONTROL OF INFLAMMATION
  od: Altonsy, Mohammed O., i dr.
  Izdano: (2014)
• Long-Acting β(2)-Adrenoceptor Agonists Enhance Glucocorticoid Receptor (GR)–Mediated Transcription by Gene-Specific Mechanisms Rather Than Generic Effects via GR
  od: Rider, Christopher F., i dr.
  Izdano: (2018)