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TCPTP-deficiency in muscle does not alter insulin signalling and glucose homeostasis
AIMS/HYPOTHESIS: Insulin activates the insulin receptor (IR) protein tyrosine kinase and downstream phosphatidylinositol-3-kinase (PI3K)/Akt signalling in muscle to promote glucose uptake. The IR can serve as a substrate for the protein tyrosine phosphatases (PTP) 1B and TCPTP, which share a strikin...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Diabetologia |
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| Κύριοι συγγραφείς: | , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2011
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5057388/ https://ncbi.nlm.nih.gov/pubmed/22124607 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00125-011-2386-z |
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