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Y-family DNA polymerase-independent gap-filling translesion synthesis across aristolochic acid-derived adenine adducts in mouse cells
Translesion DNA synthesis (TLS) operates when replicative polymerases are blocked by DNA lesions. To investigate the mechanism of mammalian TLS, we employed a plasmid bearing a single 7-(deoxyadenosine-N(6)-yl)-aristolactam I (dA-AL-I) adduct, which is generated by the human carcinogen, aristolochic...
Tallennettuna:
| Julkaisussa: | DNA Repair (Amst) |
|---|---|
| Päätekijät: | , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2016
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5048550/ https://ncbi.nlm.nih.gov/pubmed/27497692 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.dnarep.2016.07.003 |
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