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Y-family DNA polymerase-independent gap-filling translesion synthesis across aristolochic acid-derived adenine adducts in mouse cells

Translesion DNA synthesis (TLS) operates when replicative polymerases are blocked by DNA lesions. To investigate the mechanism of mammalian TLS, we employed a plasmid bearing a single 7-(deoxyadenosine-N(6)-yl)-aristolactam I (dA-AL-I) adduct, which is generated by the human carcinogen, aristolochic...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:DNA Repair (Amst)
Päätekijät: Hashimoto, Keiji, Bonala, Radha, Johnson, Francis, Grollman, Arthur P., Moriya, Masaaki
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2016
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5048550/
https://ncbi.nlm.nih.gov/pubmed/27497692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.dnarep.2016.07.003
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