Deposition of beta/A4 immunoreactivity and neuronal pathology in transgenic mice expressing the carboxyl-terminal fragment of the Alzheimer amyloid precursor in the brain.

समान संसाधन

• Brain transplants of cells expressing the carboxyl-terminal fragment of the Alzheimer amyloid protein precursor cause specific neuropathology in vivo.
  द्वारा: Neve, R L, और अन्य
  प्रकाशित: (1992)
• Extracellular vesicles: where the amyloid precursor protein carboxyl‐terminal fragments accumulate and amyloid‐β oligomerizes
  द्वारा: Pérez‐González, Rocío, और अन्य
  प्रकाशित: (2020)
• The neurotoxic carboxy-terminal fragment of the Alzheimer amyloid precursor binds specifically to a neuronal cell surface molecule: pH dependence of the neurotoxicity and the binding
  द्वारा: Kozlowski, MR, और अन्य
  प्रकाशित: (1992)
• Carboxyl-terminal fragments of beta-amyloid precursor protein bind to microtubules and the associated protein tau.
  द्वारा: Islam, K., और अन्य
  प्रकाशित: (1997)
• Lysosomal dysfunction in the brain of a mouse model with intraneuronal accumulation of carboxyl terminal fragments of the amyloid precursor protein
  द्वारा: Kaur, Gurjinder, और अन्य
  प्रकाशित: (2016)