Deposition of beta/A4 immunoreactivity and neuronal pathology in transgenic mice expressing the carboxyl-terminal fragment of the Alzheimer amyloid precursor in the brain.

Antzeko izenburuak

• Brain transplants of cells expressing the carboxyl-terminal fragment of the Alzheimer amyloid protein precursor cause specific neuropathology in vivo.
  nork: Neve, R L, et al.
  Argitaratua: (1992)
• Extracellular vesicles: where the amyloid precursor protein carboxyl‐terminal fragments accumulate and amyloid‐β oligomerizes
  nork: Pérez‐González, Rocío, et al.
  Argitaratua: (2020)
• The neurotoxic carboxy-terminal fragment of the Alzheimer amyloid precursor binds specifically to a neuronal cell surface molecule: pH dependence of the neurotoxicity and the binding
  nork: Kozlowski, MR, et al.
  Argitaratua: (1992)
• Carboxyl-terminal fragments of beta-amyloid precursor protein bind to microtubules and the associated protein tau.
  nork: Islam, K., et al.
  Argitaratua: (1997)
• Lysosomal dysfunction in the brain of a mouse model with intraneuronal accumulation of carboxyl terminal fragments of the amyloid precursor protein
  nork: Kaur, Gurjinder, et al.
  Argitaratua: (2016)