ロード中...
A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses
Cancer therapy targets malignant cells that are surrounded by a diverse complement of nonmalignant stromal cells. Therapy-induced damage of normal cells can alter the tumor microenvironment, causing cellular senescence and activating cancer-promoting inflammation. However, how these damage responses...
保存先:
| 出版年: | Genes Dev |
|---|---|
| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Cold Spring Harbor Laboratory Press
2016
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5024680/ https://ncbi.nlm.nih.gov/pubmed/27566778 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.284851.116 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|