ロード中...
Amyloid β oligomers elicit mitochondrial transport defects and fragmentation in a time-dependent and pathway-specific manner
Small oligomeric forms of amyloid-β (Aβ) are believed to be the culprit for declined brain functions in AD in part through their impairment of neuronal trafficking and synaptic functions. However, the precise cellular actions of Aβ oligomers and underlying mechanisms in neurons remain to be fully de...
保存先:
| 出版年: | Mol Brain |
|---|---|
| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2016
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4989350/ https://ncbi.nlm.nih.gov/pubmed/27535553 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-016-0261-z |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|